Autoimmune disease that may become complicated

Scleroderma is a chronic, autoimmune, rheumatic connective tissue disease.

Chronic illness can be serious. The symptoms of scleroderma vary greatly from person to person and the effects of scleroderma can range from mild to life-threatening. The seriousness will depend on the parts of the body that are affected and the extent to which they affect. A milder case can become more serious if not treated properly. The early and appropriate diagnosis and treatment by qualified medical practitioners can minimise the symptoms of scleroderma and reduce the chances of irreversible damage.

Scleroderma: how is it diagnosed?

The diagnostic process may require consultation with a rheumatologist (arthritis specialist) and/or dermatologist (skin specialist) and requires blood tests and numerous other tests depending on the organs that are affected.

Who develops scleroderma and when?

About one third of people with scleroderma have the systemic form of scleroderma. Because scleroderma presents with symptoms similar to other forms of scleroderma, it can be autoimmune diseasesdiagnosis is difficult. There are many undiagnosed or half-diagnosed cases.

Localised scleroderma is more common in children, while systemic scleroderma is more common in adults.. In general, female patients outnumber male patients about 4-to-1. Factors other than a person's sex, such as race and ethnicity, may influence the risk of scleroderma, the age of onset, and the pattern or severity of internal organ involvement. The reasons for this are unclear. Although scleroderma is not directly inherited, some scientists feel that there is a slight predisposition in families with a history of rheumatic diseases.

However, the scleroderma can develop in any age group, from children to the elderly, although its occurrence is most common between the ages of 25 and 55.. When it says "usually" or "for the most part," the reader should understand that there may be variations. Many patients are alarmed when they read medical information that seems to contradict their own experiences, and conclude that what has happened to them is not supposed to have happened to them. There are many exceptions to the rules in scleroderma, perhaps more than in other diseases. Each case is different, and information should be discussed with one's own doctor.

Scleroderma. Causes

The exact cause or causes of scleroderma are still unknown, but scientists and medical researchers are working to reach some conclusions. It is known that scleroderma involves an overproduction of collagen.

Is scleroderma genetic?

Most patients do not have relatives with scleroderma and their children do not inherit scleroderma. Research indicates that there is a susceptibility gene, which increases the likelihood of getting scleroderma, but does not by itself cause the disease.

What is the treatment for scleroderma?

Currently, there is no cure for scleroderma, but there are many treatments available to help particular symptoms. For example, heartburn can be controlled by medications called proton pump inhibitors (PPIs) or medications to improve bowel movement and heartburn. Some treatments are aimed at decreasing the activity of the immune system. People with mild disease may not need medication at all and sometimes people may not take treatment when their scleroderma is no longer active. Because there is so much variation from person to person, there is a wide variation in the treatments prescribed..

Scleroderma. Biological treatment

We protocolise a personalised treatment in several phases where we combine all the techniques of Biological Medicine and, if necessary, Conventional Medicine. If necessary, we prescribe a natural symptomatic treatment. The first thing we do is a functional regulation, a drainage of toxinsand, if necessary, the chelation therapy to eliminate heavy metals, together with intestinal cleansing. After about 4 weeks, we will continue with the treatment of bacterial and/or viral and/or fungal infections. After a period of at least 8 weeks, we will have a regulation of the immune system and a deactivation of genetic cargoes that have been activated in the patient to develop autoimmune disease.